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When a cell over-expresses NNMT, it does two costly things at once: it burns through methyl-donor capacity, and it diverts nicotinamide away from NAD+ regeneration into an inert metabolite (1-methylnicotinamide) that gets excreted
BPC 157 is thought to stimulate the production of nitric oxide, which in turn increases healing by improving blood circulation to the damaged tissues
A further study investigating tendon-to-bone healing following the transection and detachment of the Achilles tendon in rats went on to reveal that BPC 157 is also capable of promoting tendon-to-bone healing despite the presence of corticosteroids (Krivic et al
Together they provide broader tissue repair mechanism coverage
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