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This domain of Ric1 is bound to the nucleotide-binding domain of Rab6 (Fig

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The researchers determined breast cancer incidence as the primary study outcome

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308 Rather than harboring stable resistance mutations, DTP cells adopt a slow-cycling or quasiquiescent phenotype driven by extensive epigenetic remodeling, transcriptional plasticity and metabolic reprogramming, which collectively constitute core maintenance programs that stabilize the DTP state under therapeutic pressure