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The FTD-like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism
Both peptides also show overlapping anti-inflammatory signals in separate studies, with each associated with reduced pro-inflammatory cytokines such as IL-6 and TNF-alpha in animal models
C.Fuentes-FayosA
That is exactly why we treat this as a supervised, individualized therapy with lab monitoring rather than a guaranteed outcome
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