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It is important to note that if ED develops or worsens during GLP-1 therapy, this is more likely related to the underlying conditions being treated (diabetes, obesity, cardiovascular disease) or other concurrent factors rather than the medication itself
It was first identified by researchers at Harvard in 1987, when scientists Mojsov, Weir, and Habener discovered it was produced in the gut and powerfully triggered insulin release
Yunna C, Mengru H, Lei W, Weidong C
It is not a single-pathway molecule
These medicines work by mimicking the action of the naturally occurring GLP-1 hormone, which stimulates insulin secretion in response to food intake, suppresses glucagon release, slows gastric emptying, and promotes satiety
