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Pharmacologic inhibition of CXCR4 has been shown to rejuvenate T-cell function and restore sensitivity to ICB
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Beta blockers suppress cardiac output and inhibit mediated vasodilation, both effects could result in reduced RBF
21 In fibroblasts 3T3/4A, GSTP1/JNK interaction occurring in the absence of stress stimuli also inhibits JNK activity and c-Jun phosphorylation
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