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S1AS1C), suggesting that even minimal production of GSH can maintain homeostatic processes in the body
Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways
The authors acknowledge this limitation
A pharmacist suitability assessment is required
Antonov, N
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