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Beyond its cytotoxic effects, Paclitaxel has shown immune-modulatory properties, specifically reprogramming TAMs from a tumor-promoting M2 phenotype to an antitumor M1 phenotype through TLR4 (Toll-like receptor 4) signaling

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This allows it to cross cell membranes intact, where it is converted into active glutathione inside the cell

In addition to the tumor matrix, a complex interplay between immune cells and tumor amino acid metabolism is emerging