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10.1046/j.1365-2958.1999.01271.x Mol
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These results suggest that HSV-1 can modulate this pathway at different time-points after infection by interfering with apoptotic signaling events to favor its replication at early times in the replication cycle and after supporting neuronal survival for its persistence in a latent state
it slows down how quickly starches turn to glucose, therefore creating a massive reduction in the glucose spike of a meal
The clinical course of MS is generally indolent and metastasis are rarely seen
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