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Studies have shown that in the HF mouse of myocardial infarction models, the depletion of neutrophils through antibody-mediated or genetic methods can halt the progression of HF, especially the left ventricular remodeling and fibrosis progression in the middle and late stages of HF (135), further indicating that neutrophils are a potential therapeutic target for lowering myocardial damage and preventing HF (159, 160)

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10.3390/antiox5030030 55 LiuB.MaoQ.CaoM.XieL

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