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KLOW dosage in research contexts is determined by the investigator's experimental design and the target concentration for each component
KMT2C deficiency simultaneously enhances fatty acid biosynthesis and HRAS palmitoylation, activating the MAPK/ERK pathway and stabilizing resistance phenotypes
Of note, treatment with the neutralizing IGF-IR monoclonal antibody is incapable of overcoming resistance to hormone deprivation or antiestrogen therapy, due to the compensation by InsR [107]
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