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Here we investigate the effects of liraglutide, a GLP-1 receptor agonist, in two well characterised transgenic mouse models of ALS (SOD1 G93A and TDP-43 Q331K ) to determine if liraglutide could be a potential treatment in ALS patients

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Functional analyses of this mutant showed that it is also strongly impaired for cooperation with BRCA2-DSS1 in RAD51 presynaptic filament assembly, with RAD51 in synaptic complex formation and in DNA strand exchange and is also incapable of protecting DNA against the nuclease activity of MRN

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