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Lentz et al, 1996) investigations that suggest a causal relationship between elevated homocysteine levels and vascular spasm and thrombosis and with cross-sectional studies, which suggest that elevated homocysteine levels contributed to acute fatal thromboembolic events (Alfthan et al, 1997

These side effects can usually be managed and improved over time

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In process (9), GSH acts as the precursor that allows HMs to bind to their thiol (-SH) group to form PC, catalyzed by the enzyme phytochelatin synthase (PCS)

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