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An increased activation of IDO and its subsequent enzyme kynurenine monooxygenase by proinflammatory cytokines, moreover, leads to an enhanced production of quinolinic acid, a strong agonist of the glutamatergic N -methyl-D-aspartate receptor
This design increases its half-life from minutes to several hours, allowing for greater receptor activation in tissues such as muscle and tendon
Rahman, Inflammation and the Regulation of Glutathione Level in Lung Epithelial Cells, Antioxid
Mitochondrial dysfunction also contributes to sepsis-induced myopathy (SIM)
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