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There, it activates pro-apoptotic proteins such as BAX and cleaved Caspase-3, initiating the apoptosis cascade exclusively in the senescent cell [2]
This was performed over the course of almost three months with regular treatments every two weeks
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Proposed Mechanisms Preventing GABA receptor downregulation Counteracting compensatory NMDA glutamate receptor sensitization Stabilizing benzodiazepine receptor configuration (preventing shift to inverse agonist state) Upregulating GABAergic neurotransmission through pathways distinct from benzodiazepine action itself This tolerance prevention has profound clinical implications
