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Acknowledgements We wish to thank our collaborators (Drs
10.1371/journal.pone.0232053 53
(Abbreviations used in this paper along with their complete names are listed in Glossary.) Figure 1 An important theme in interpreting vitamin B 12 deficiency is the remethylation block hypothesis: Vitamin B 12 deficiency results in hyperhomocysteinemia because Hcy remethylation is blocked (Selhub et al., 2007)
It appears at least at mouse islet GLP-1R that pGLP-1 would act with classical incretin function to promote insulin release, although whether this is the primary function in the platypus remains to be proven
Limitations of current therapeutic strategies Current clinical management of RIRI primarily relies on antioxidant agentssuch as N-acetylcysteine and vitamins E/Canti-inflammatory drugs including non-steroidal anti-inflammatory agents (NSAIDs), and nephroprotective compounds like amino acids and indomethacin, often supplemented by supportive measures such as fluid resuscitation and extracorporeal blood purification
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