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Activation of this circuit in obese mice reduces food intake and improves glucose tolerance by stimulating insulin secretion via inhibition of GABA-input-dependent sympathetic nervous activity in the paraventricular hypothalamus
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In addition to delivering meaningful weight loss, mGIPR-Ab/GLP-1 also provides metabolic benefits in downstream peripheral tissues
This distinction proves important as appropriate inflammatory response is necessary for healing, but excessive or prolonged inflammation impairs recovery
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