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Increased levels of NAD+ during an infection increase oxidative phosphorylation in macrophages, which makes them better able to neutralize the organism causing the infection

Gastrointestinal and tendon/ligament research: BPC-157 has the most consistent preclinical signal here, including reported reductions in gastric ulcer surface area and accelerated tendon healing in rodent models

These findings provoke interesting primary questions as to the possible origins of PD-associated alpha-synuclein misfolding from within autonomic nerve fibers of the gut

SGLT-2 inhibitors: the gap seems to be leveling off

Cd40 signaling in Graves disease is mediated through canonical and noncanonical thyroidal nuclear factor B activation