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It signals the body to preserve muscle tissue during caloric deficit, improves insulin sensitivity beyond what the medication achieves alone, increases metabolic rate, and shapes the body in ways that fat loss alone cannot achieve

doi: 10.2215/CJN.02960906 [13] Boucher BJ

shown are the GLP-1-bound (orange) and TT-OAD2 bound (blue) GLP-1R

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Transamination can thus be linked to deamination, effectively allowing nitrogen from the amine groups of amino acids to be removed, via glutamate as an intermediate, and finally excreted from the body in the form of urea