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Mechanistically, melatonin acted independently of canonical melatonin receptors and instead reprogrammed ILC2 metabolism toward pentose phosphate pathway activity, enhancing NADPH generation and NRF2-dependent glutathione accumulation
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For both the wild-type and hepsin / mice treated with 2APB, the survival rate increased significantly compared with the vehicle control group at 72 h post-APAP administration (Fig
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